Skip to main content

What's Wobble in EP Study


 In electrophysiology (EP) study), the term "wobble" usually refers to:


πŸ‘‰ Variation in the cycle length of a tachycardia from beat to beat — especially during AV nodal reentrant tachycardia (AVNRT) or other reentrant arrhythmias.


In reentrant circuits, the conduction time around the circuit may fluctuate slightly due to changes in refractoriness or conduction velocity.


This produces small irregularity in the tachycardia cycle length (not perfectly fixed as in atrial flutter).


On intracardiac recordings, this is seen as beat-to-beat variation in interval timing (wobbling).



πŸ”Ή Clinical importance:


Helps differentiate reentrant tachycardias (which may “wobble”) from automatic tachycardias (which are usually more regular).


In AV nodal physiology, wobble of the AH interval or tachycardia cycle length during induction/termination gives clues to mechanism.



πŸ”Ή Example 1: AH interval wobble in AVNRT


During AV nodal reentrant tachycardia, the conduction through the AV node (AH interval) can vary slightly beat-to-beat.


Suppose AH = 120 ms on one beat, then 130 ms, then 125 ms → this variation is wobble.


On the intracardiac recording:


A → H interval doesn’t stay flat.


But H → V interval remains constant.



This supports reentry via AV node rather than automatic focus.




---


πŸ”Ή Example 2: Tachycardia cycle length wobble


A patient has SVT at ~350 ms cycle length.


You measure: 348 ms → 354 ms → 349 ms → 352 ms.


That small, irregular “breathing” of cycle length = wobble.


It suggests a reentrant mechanism (AVNRT or accessory pathway) rather than atrial tachycardia (which tends to be more stable if automatic).




---


πŸ”Ή Example 3: Why flutter is “fixed” and doesn’t wobble


Atrial flutter circuit is large and anatomical (cavo-tricuspid isthmus).


Cycle length stays very regular (e.g., exactly 240 ms each beat).


No wobble → favors macroreentry like flutter.




---


✅ Take-home pearl:


Wobble = small beat-to-beat variability in conduction intervals or tachycardia cycle length on EP tracings.


Supports reentry as mechanism.


Absent wobble (very fixed cycle length) → favors automatic tachycardia or atrial flutter.



Comments

Popular posts from this blog

STEMI ECG Criteria and Universal Definition of MI

  STEMI ECG Criteria and the Universal Definition of Myocardial Infarction: A Complete Guide for Clinicians Early and accurate diagnosis of acute myocardial infarction (AMI) remains the cornerstone of reducing morbidity and mortality in patients presenting with chest pain. Among all forms of acute coronary syndromes (ACS), ST-elevation myocardial infarction (STEMI) represents the most time-sensitive emergency, requiring immediate reperfusion therapy. This article provides a clinically relevant summary of the STEMI ECG criteria and the Universal Definition of Myocardial Infarction (UDMI), based on the latest consensus guidelines from the ESC, ACC, AHA, and WHF. --- 1. Understanding STEMI: Why Accurate ECG Interpretation Matters A 12-lead ECG remains the first and most critical diagnostic test when evaluating suspected myocardial infarction. STEMI is identified when there is evidence of acute coronary artery occlusion, producing transmural ischemia and characteristic ST-segment eleva...

2025 AHA/ACC Hypertension Guidelines Key points

  2025 AHA/ACC Hypertension Guidelines Explained: A Clear Summary for Clinicians and Students Hypertension remains one of the most significant contributors to cardiovascular morbidity and mortality worldwide. With continual refinement of evidence and risk-based strategies, the 2025 AHA/ACC Hypertension Guidelines bring an updated, practical approach that clinicians can use in daily practice. To make learning easier, I’ve created a clean and modern infographic summarizing all major recommendations. You can download it below and use it for study, teaching, or clinical reference. Download Infographic (PNG): 2025 Hypertension Guideline Infographic This post breaks down the key points from the guidelines and complements the infographic for a complete understanding. --- BP Categories: Understanding the Updated Thresholds The guidelines maintain the well-established classification of blood pressure: Normal: <120 / <80 Elevated: 120–129 / <80 Stage 1 Hypertension: 130–139 and/or 8...

π˜Όπ™£π™©π™žπ™˜π™€π™–π™œπ™ͺπ™‘π™–π™©π™žπ™€π™£ π˜Όπ™›π™©π™šπ™§ π™Žπ™©π™§π™€π™ π™š

 π˜Όπ™£π™©π™žπ™˜π™€π™–π™œπ™ͺπ™‘π™–π™©π™žπ™€π™£ π˜Όπ™›π™©π™šπ™§ π™Žπ™©π™§π™€π™ π™š in  Patient with AF and acute IS/TIA European Heart Association Guideline recommends: • 1 days after TIA • 3 days after mild stroke • 6 days after moderate stroke • 12 days after severe stroke Early anticoagulation can decrease a risk of recurrent stroke and embolic events but may increase a risk of secondary hemorrhagic transformation of brain infarcts.  The 1-3-6-12-day rule is a known consensus with graded increase in delay of anticoagulation between 1 and 12 days after onset of ischemic stroke or transient ischemic attack(TIA), according to neurological severity based on European expert opinions. However, this rule might be somewhat later than currently used in a real-world practical setting.