VT vs SVT with Aberrancy – ECG Criteria for Accurate Diagnosis
Wide complex tachycardia (WCT) is VT unless proven otherwise. Correct differentiation between ventricular tachycardia (VT) and supraventricular tachycardia (SVT) with aberrancy is critical because management differs significantly and misdiagnosis can be dangerous.
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1. Basic Definitions
Ventricular Tachycardia (VT)
A tachycardia originating below the His bundle (ventricular myocardium or Purkinje system), usually with wide QRS ≥ 120 ms.
SVT with Aberrancy
A supraventricular rhythm conducted through abnormal ventricular conduction (e.g., bundle branch block or accessory pathway), producing a wide QRS.
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2. Why It Matters
Treating VT as SVT (e.g., giving AV nodal blockers in unstable VT) can cause hemodynamic collapse.
Treating SVT as VT is generally safer (e.g., amiodarone).
In structural heart disease, ischemic heart disease, or prior MI → assume VT.
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Stepwise ECG Approach to Wide Complex Tachycardia
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Step 1: Clinical Clues
Suggests VT:
Age > 35 years
Structural heart disease
Previous MI
Cardiomyopathy
History of ventricular arrhythmia
Suggests SVT:
Known SVT history
Young patient without structural disease
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Step 2: AV Dissociation (Most Specific Sign of VT)
Findings:
P waves independent of QRS
Capture beats
Fusion beats
If AV dissociation present → VT until proven otherwise
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Step 3: QRS Duration
QRS ≥ 140 ms (RBBB morphology) → favors VT
QRS ≥ 160 ms (LBBB morphology) → favors VT
Very wide QRS (> 180 ms) → strongly VT
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Step 4: Concordance in Chest Leads
Definition: All precordial leads either entirely positive or entirely negative
Positive concordance → VT
Negative concordance → VT
Concordance is highly specific for VT.
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Step 5: Brugada Criteria
Developed by Pedro Brugada.
Four-step algorithm:
1. Absence of RS complex in all precordial leads → VT
2. RS interval > 100 ms in any precordial lead → VT
3. AV dissociation present → VT
4. Morphologic criteria for VT in V1–V2 and V6
If any step positive → VT
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Step 6: Vereckei (aVR) Algorithm
Developed by LΓ‘szlΓ³ Vereckei.
Focuses mainly on lead aVR:
Initial R wave in aVR → VT
Initial r or q > 40 ms in aVR → VT
Notching on downstroke of QRS in aVR → VT
Vi/Vt ratio ≤ 1 → VT
Useful when bundle branch morphology is unclear.
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Step 7: Morphologic Criteria
If RBBB Pattern in V1
Suggests VT if:
Monophasic R
qR pattern
Broad R (> 40 ms)
R/S ratio < 1 in V6
Suggests SVT if:
rSR' typical pattern
Narrow initial r wave
If LBBB Pattern in V1
Suggests VT if:
Broad initial R in V1
Notched S in V1
Q wave in V6
Suggests SVT if:
Typical LBBB morphology
Narrow initial r wave
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Comparative Table
Feature VT SVT with Aberrancy
AV dissociation Present Absent
Capture/Fusion beats Present Absent
QRS width Very wide Moderately wide
Concordance Often present Rare
Structural heart disease Common Less common
Response to adenosine No termination May terminate
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Special Situations
1. Pre-excited SVT (WPW with AF)
Irregular wide tachycardia
Very fast ventricular rate
Avoid AV nodal blockers
2. Fascicular VT
Relatively narrow QRS
RBBB morphology
Responds to verapamil
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Practical Emergency Rule
If unsure → treat as VT
Unstable → immediate synchronized cardioversion
Stable → amiodarone or procainamide
Avoid verapamil in undifferentiated WCT (risk of collapse in VT).
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Key Take-Home Points
AV dissociation is the most specific ECG sign of VT.
Concordance strongly favors VT.
Use Brugada or Vereckei algorithm systematically.
Wide complex tachycardia in structural heart disease = VT until proven otherwise.
When in doubt, manage as VT.

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