Skip to main content

π€π§πšπ­π¨π¦π’πœ 𝐚𝐩𝐩𝐫𝐨𝐚𝐜𝐑 𝐭𝐨 𝐬π₯𝐨𝐰 𝐩𝐚𝐭𝐑𝐰𝐚𝐲 πšπ›π₯𝐚𝐭𝐒𝐨𝐧 𝐒𝐧 𝐀𝐕𝐍𝐑𝐓.



π€π§πšπ­π¨π¦π’πœ 𝐚𝐩𝐩𝐫𝐨𝐚𝐜𝐑 𝐭𝐨 𝐬π₯𝐨𝐰 𝐩𝐚𝐭𝐑𝐰𝐚𝐲 πšπ›π₯𝐚𝐭𝐒𝐨𝐧 𝐒𝐧 𝐀𝐕𝐍𝐑𝐓. 

The septal aspect of the tricuspid annulus from the CS ostium to the His bundle is divided in posterior (Post), mid (Mid), and anterior (Ant) sections (from the RAO view). The ablation catheter is initially placed in the posterior section and RF applications are started here. Successful ablation may require progressive advancement of the ablation catheter to the mid and then to the anterior sites with increased risk of AV block in the more superior and anterior sites.


The anatomic method targets the slow pathway region in the right atrial posteroseptum, guided by fixed landmarks rather than detailed electrogram criteria.


Key Anatomical Landmarks


1. Triangle of Koch — the critical map:


Apex: His bundle region


Base: Ostium of the coronary sinus (CS)


Septal leaflet of the tricuspid valve (TV) forming the third border




2. Slow pathway zone:


Located inferior and posterior to the His region


Typically around the posterior third of the triangle near the CS ostium





Target Site (Preferred Ablation Region)


Inferior/posterior part of the Triangle of Koch, between:


the CS ostium,


the tricuspid annulus, and


just inside the Eustachian ridge area.



This region often shows A < V electrograms but EGM quality is not mandatory in the pure anatomic technique.



Technical Steps


1. Place His catheter to mark the apex (avoid ablation too close).



2. Engage CS catheter to define the base.



3. Position the ablation catheter just anterior (septal) to the CS ostium, sliding slightly upward if needed.



4. Deliver RF energy at 30–50W (temp-limited if using older generators).



5. Watch for:


Accelerated junctional rhythm → indicates correct slow pathway modification.


Avoid prolonged AV block—stop if VA dissociation or PR prolongation occurs.





Endpoints


Non-inducibility of AVNRT (with/without isoproterenol).


Preserved fast pathway conduction (normal AV conduction).


Occasional single echo is acceptable if no sustained tachycardia.



Advantages of Anatomic Approach


Faster


Less mapping-dependent


Very high success (≈95%)


Extremely low AV block risk when targeting the posteroinferior zone





Comments

Popular posts from this blog

π˜Όπ™£π™©π™žπ™˜π™€π™–π™œπ™ͺπ™‘π™–π™©π™žπ™€π™£ π˜Όπ™›π™©π™šπ™§ π™Žπ™©π™§π™€π™ π™š

 π˜Όπ™£π™©π™žπ™˜π™€π™–π™œπ™ͺπ™‘π™–π™©π™žπ™€π™£ π˜Όπ™›π™©π™šπ™§ π™Žπ™©π™§π™€π™ π™š in  Patient with AF and acute IS/TIA European Heart Association Guideline recommends: • 1 days after TIA • 3 days after mild stroke • 6 days after moderate stroke • 12 days after severe stroke Early anticoagulation can decrease a risk of recurrent stroke and embolic events but may increase a risk of secondary hemorrhagic transformation of brain infarcts.  The 1-3-6-12-day rule is a known consensus with graded increase in delay of anticoagulation between 1 and 12 days after onset of ischemic stroke or transient ischemic attack(TIA), according to neurological severity based on European expert opinions. However, this rule might be somewhat later than currently used in a real-world practical setting.

Acute Treatment of Hyperkalemia

Acute Treatment of Hyperkalemia – A Practical, Bedside-Oriented Guide Hyperkalemia is a potentially life-threatening electrolyte abnormality that demands prompt recognition and decisive management. The danger lies not only in the absolute potassium value but in its effects on cardiac conduction, which can rapidly progress to fatal arrhythmias. Acute treatment focuses on three parallel goals: stabilizing the cardiac membrane, shifting potassium into cells, and removing excess potassium from the body. Understanding this stepwise approach helps clinicians act quickly and rationally in emergency settings. Why Hyperkalemia Is Dangerous Potassium plays a key role in maintaining the resting membrane potential of cardiac myocytes. Elevated serum potassium reduces the transmembrane gradient, leading to slowed conduction, ECG changes, ventricular arrhythmias, and asystole. Importantly, ECG changes do not always correlate with potassium levels, so treatment decisions should be based on clinical c...

2025 AHA/ACC Hypertension Guidelines Key points

  2025 AHA/ACC Hypertension Guidelines Explained: A Clear Summary for Clinicians and Students Hypertension remains one of the most significant contributors to cardiovascular morbidity and mortality worldwide. With continual refinement of evidence and risk-based strategies, the 2025 AHA/ACC Hypertension Guidelines bring an updated, practical approach that clinicians can use in daily practice. To make learning easier, I’ve created a clean and modern infographic summarizing all major recommendations. You can download it below and use it for study, teaching, or clinical reference. Download Infographic (PNG): 2025 Hypertension Guideline Infographic This post breaks down the key points from the guidelines and complements the infographic for a complete understanding. --- BP Categories: Understanding the Updated Thresholds The guidelines maintain the well-established classification of blood pressure: Normal: <120 / <80 Elevated: 120–129 / <80 Stage 1 Hypertension: 130–139 and/or 8...